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Skin Aging
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Throughout
life and thins the
skin ages due to lack of repair.
At age 45 is clearly visible
signs of aging evident.
At age 75 the bad image
the main complication is
usually with white
hair, wrinkles, warts, rashes ....
Beyond 75 years it
can only be expected that
cancer as a farewell present.
Skin Aging The Sun is the main enemy of the
skin. The little light that penetrates the epidermis to the deeper
layer, destroys stem cells. Gradually, over the years, the light
undermines the stratum germinativum of the epidermis. As a result of the
loss of many stem cells, the epidermis decreases in thickness. The horny
layer, less compact, it becomes rough with scales that are raised and
off easily. The skin loses its own clear radiance of youth. By reducing
their capacity for reflection, the light penetrates more and deeper into
the epidermis. This vicious cycle is perpetuated and is growing every
day. At 75 years, the epidermis is so thin that lets plenty of sunlight.
Now the light is no longer reflected in the stratum corneum but also in
the dermis. Due to the reflection of light on the blood vessels in the
dermis, the skin looks red. Epidermis: In areas of the epidermis with
the highest number of casualties keratinocyte stem cells, there is a
decrease of keratinocytes. 1 The proportion epidermal melanocyte per 20
keratinocytes, typical of youth, to move toward a relative increase of
melanocytes. Abundant sunlight reaching the stratum germinativum,
overstimulated melanocytic stem cells. Actively divide and cause
hyperplasia of keratinocytes. This is displayed as dark spot and touch
warty, as actinic keratosis, senile seborrheic keratosis and freckles.
Dennis: As stem cells die in the dermis, it becomes thinner and loses
consistency. The skin is partially folded over itself and wrinkles
appear. Hypodermis: The thinning of the fat layer produces a loss of
volume. Disappears "volume visage" characteristic of youth.
The aging of the skin may continue and be even
more complicated than simple aging skin. When the cell gets older and no
longer useful for their role in tissue, communicates to his mother and
sisters surrounding her intention to commit suicide. Tell all the cells
around them, exposing suicide proteins outside the cell membrane. The
stem cell that originated also attends the funeral. You have to fill the
gap that will leave your daughter to die. But if the stem cell that
originated not attend because he has spent his division number 60 and
has died, the ad remains exposed in a long time outside the cell. This
is a dangerous virus because there are many waiting to get inside the
cell.
Apoptosis or cell suicide announced, is a
programmed death, and unfortunately sometimes too long announced. The
announcement is, at times, very well received by viruses that have the
intention to utilize the old cell to recycle it and give it a new life
for them. Some viruses, such as Human Papilloma Virus, can bind to these
proteins in the cell membrane to enter the cell.
The viruses infecting these cells become
moribund and bereft of recycled mother cells capable of dividing
endlessly. Cells infected by these viruses work for them as cancer
cells. In each cell division creates a new cancerous and cancerous cells
in each of them has doubled and hosted a new oncogenic virus. The aging
of the skin is justified by a growing number of orphans aged cells. Skin
cancer most frequently in their presentations, such as squamous cell
carcinoma, basal cell carcinoma and malignant melanoma appears in aged
skin, especially hit by the sun, suggesting that beyond the division
number 60 there is no life. There is only cell death or cancer death.
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